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Many anticancer agents induce apoptosis via activation of the MAP kinase pathway, specifically via JNK and p38 (Dav is, 2000
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Sido et al, 1998), the increase in -GCS in mucosa from TNBS-treated rats can be interpreted as an adaptive response to control the consequences of the stress induced by TNBS as -GCS-HS is known to be upregulated in response to divergent stressful conditions including oxidative stress or GSH depletion (Morales et al, 1997, 1998
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